prism version 3.00 for windows software (GraphPad Software Inc)
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90
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GraphPad Software Inc
prism version 3.00 for windows software
Prism Version 3.00 For Windows Software, supplied by GraphPad Software Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/prism+windows%2C+version+3%2E00/prism+software/pm37330403-179-28-23
Average 90 stars, based on 1 article reviews
Prism Version 3.00 For Windows Software, supplied by GraphPad Software Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/prism+windows%2C+version+3%2E00/prism+software/pm37330403-179-28-23
Average 90 stars, based on 1 article reviews
prism version 3.00 for windows software - by Bioz Stars,
2026-09
90/100 stars
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other:Article Title: Tumor necrosis factor-alpha up-regulates the expression of CCL2 and adhesion molecules of human proximal tubular epithelial cells through MAPK signaling pathways. Article Snippet: Both circulating and urinary tumor necrosis factor (TNF)-a levels have been shown to increase in inflammatory chronic kidney diseases and TNF-a can induce secretion of other inflammatory mediators from many cell types.. Chemokine, mononuclear chemoattractant protein-1 (CCL2/MCP-1), and cell surface adhesion molecules, intercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion molecule-1 (VCAM-1), in renal proximal tubular epithelial cells (PTEC) are important for promoting recruitment and adhesion of infiltrating macrophages and lymphocytes to inflamed renal tissue.. This study aimed to investigate the effect of TNF-a on the expression of these inflammation-related molecules of human PTEC and the underlying intracellular mitogen-activated protein kinase (MAPK) regulatory signaling mechanisms. Article Title: Induction of adhesion molecules upon the interaction between eosinophils and bronchial epithelial cells: involvement of p38 MAPK and NF-kappaB. Article Snippet: Eosinophils are principal effector cells of inflammation in allergic asthma, characterized by their infiltration and accumulation at inflammatory sites mediated by chemokine eotaxin, and interaction with adhesion molecules expressed on bronchial epithelial cells.. In this study, tumor necrosis factor (TNF)-α and/or the interaction of eosinophils and bronchial epithelial BEAS-2B cells were found to upregulate the cell surface expression of adhesion molecules intercellular adhesion molecule (ICAM)-1 and vascular adhesion molecule (VCAM)-1 on BEAS-2B cells, and ICAM-1 and leukocyte function-associated antigen-1 (LFA-1) on eosinophils.. Interaction of eosinophils and BEAS-2B cells could induce the release of granulocyte macrophage colony-stimulating factor (GM-CSF) and activate both p38 mitogen-activated protein kinase (MAPK) and nuclear factor (NF)-κB activities in BEAS-2B cells but only NF-κB activity in eosinophils. Article Title: Establishment and characterization of a novel primary hepatocellular carcinoma cell line with metastatic ability in vivo Article Snippet: All analyzes were performed using the statistical software Article Title: House dust mite allergen Der p 1 elevates the release of inflammatory cytokines and expression of adhesion molecules in co-culture of human eosinophils and bronchial epithelial cells. Article Snippet: All analyses were performed using the statistical software Article Title: Granulin-Epithelin Precursor Is an Oncofetal Protein Defining Hepatic Cancer Stem Cells Article Snippet: All analyzes were performed using the statistical software |